SARS-CoV-2 evades natural killer cell cytotoxic responses
Por um escritor misterioso
Last updated 20 setembro 2024
In a recent study posted to the bioRxiv* preprint server, researchers at Stanford University School of Medicine revealed the mechanism via severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) escapes natural killer (NK) cell cytotoxic reactions.
Frontiers Covid-19: Perspectives on Innate Immune Evasion
Full article: ORF6, a repressor of the MHC class I pathway: new molecular target for SARS-CoV-2 drug discovery?
Off balance: Interferons in COVID-19 lung infections - eBioMedicine
SARS-CoV-2 accessory proteins ORF7a and ORF3a use distinct mechanisms to down-regulate MHC-I surface expression
The role of NK cells in fighting the virus infection and sepsis
Natural Killer Cell Regulation of B Cell Responses in the Context of Viral Infection
The Emergence of NK Cell-Based Treatment in Cancer Immunotherapy
Frontiers HLA-C dysregulation as a possible mechanism of immune evasion in SARS-CoV-2 and other RNA-virus infections
Circulating Spike Protein Detected in Post–COVID-19 mRNA Vaccine Myocarditis
Acute SARS-CoV-2 Infection Impairs Dendritic Cell and T Cell Responses - ScienceDirect
Immunogenicity mechanism of mRNA vaccines and their limitations in promoting adaptive protection against SARS-CoV-2 [PeerJ]
Natural killer cell specificity for viral infections
SARS-CoV-2 evades natural killer cell cytotoxic responses
Natural killer cells and their exosomes in viral infections and related therapeutic approaches: where are we?, Cell Communication and Signaling
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