Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN
Por um escritor misterioso
Last updated 15 março 2025


Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling

Mitochondrial respiration contributes to the interferon gamma response in antigen-presenting cells
Free Fatty Acids Induce Apoptosis of Mammary Epithelial Cells of Ketotic Dairy Cows via the Mito-ROS/NLRP3 Signaling Pathway

Mitochondria-mediated oxidative stress during viral infection: Trends in Microbiology

Type 1 Interferons Induce Changes in Core Metabolism that Are Critical for Immune Function - ScienceDirect

Elevated type I interferon responses potentiate metabolic dysfunction, inflammation, and accelerated aging in mtDNA mutator mice

The role of autophagy in pro‐inflammatory responses of microglia activation via mitochondrial reactive oxygen species in vitro - Ye - 2017 - Journal of Neurochemistry - Wiley Online Library

IL-12 signaling pathway. IL-12 activates the Jak/STAT pathway.

Effects of mitochondrial reactive oxygen species-induced NLRP3 inflammasome activation on trichloroethylene-mediated kidney immune injury - ScienceDirect

PDF) Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling

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Summary of the biology of IL-12 [14]. The main physiological producers

IJMS, Free Full-Text
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