RVA-induced necrosome formation consisting of RIPK1, RIPK3, and MLKL
Por um escritor misterioso
Last updated 20 março 2025


The Structural Basis of Necroptotic Cell Death Signaling: Trends in Biochemical Sciences

RIP1/RIP3-regulated necroptosis as a target for multifaceted disease therapy (Review)

Bypassing drug resistance by triggering necroptosis: recent advances in mechanisms and its therapeutic exploitation in leukemia, Journal of Experimental & Clinical Cancer Research

Inhibition of MLKL-dependent necroptosis via downregulating interleukin-1R1 contributes to neuroprotection of hypoxic preconditioning in transient global cerebral ischemic rats, Journal of Neuroinflammation

Blocking necroptosis reduces inflammation and tumor incidence in a mouse model of diet-induced hepatocellular carcinoma

MLKL in cancer: more than a necroptosis regulator. - Abstract - Europe PMC

Overview of necroptotic signaling pathway. Necroptosis can be induced

Bypassing drug resistance by triggering necroptosis: recent advances in mechanisms and its therapeutic exploitation in leukemia, Journal of Experimental & Clinical Cancer Research

RIPK3 signaling and its role in the pathogenesis of cancers

From (Tool)Bench to Bedside: The Potential of Necroptosis Inhibitors

TAM Kinases Promote Necroptosis by Regulating Oligomerization of MLKL - ScienceDirect

Curcumol induces RIPK1/RIPK3 complex-dependent necroptosis via JNK1/2-ROS signaling in hepatic stellate cells - ScienceDirect

Transient knockdown of components along the RIPK1–RIPK3–MLKL–PGAM5–Drp1

Cardiomyocyte Alpha-1A Adrenergic Receptors Mitigate Postinfarct Remodeling and Mortality by Constraining Necroptosis

RIPK3 - an overview ScienceDirect Topics
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